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	<title>Heart pearls! &#187; ECG</title>
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	<link>http://www.heartpearls.com</link>
	<description>For all cardiology enthusiasts! Bonjour! Click on the title above to go to site index so that you can browse the articles!</description>
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		<title>ECG- Sgarbossa criteria- to diagnose AMI in LBBB</title>
		<link>http://www.heartpearls.com/2009/07/ecg-sgarbossa-criteria-to-diagnose-ami-in-lbbb.html</link>
		<comments>http://www.heartpearls.com/2009/07/ecg-sgarbossa-criteria-to-diagnose-ami-in-lbbb.html#comments</comments>
		<pubDate>Sat, 25 Jul 2009 18:34:41 +0000</pubDate>
		<dc:creator>Dr Jayachandran Thejus MD</dc:creator>
				<category><![CDATA[ECG]]></category>

		<guid isPermaLink="false">http://www.heartpearls.com/?p=474</guid>
		<description><![CDATA[5 points for concordant 1 mm ST elevation. (any lead) 3 points for concordant 1 mm ST depression in v1 to v3 2 points for discordant 5 mm ST elevation. (any lead) Score of 3 predicts AMI.]]></description>
			<content:encoded><![CDATA[<ol>
<li>5 points for concordant 1 mm ST elevation. (any lead)</li>
<li>3 points for concordant 1 mm ST depression in v1 to v3</li>
<li>2 points for discordant 5 mm ST elevation. (any lead)</li>
</ol>
<ul>
<li>Score of 3 predicts AMI.</li>
</ul>
]]></content:encoded>
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		</item>
		<item>
		<title>ECG- AV block</title>
		<link>http://www.heartpearls.com/2009/07/ecg-av-block.html</link>
		<comments>http://www.heartpearls.com/2009/07/ecg-av-block.html#comments</comments>
		<pubDate>Fri, 24 Jul 2009 12:40:56 +0000</pubDate>
		<dc:creator>Dr Jayachandran Thejus MD</dc:creator>
				<category><![CDATA[ECG]]></category>

		<guid isPermaLink="false">http://www.heartpearls.com/?p=467</guid>
		<description><![CDATA[Third degree AV block May be isoarrhythmic- similar P and QRS rates. Acute- At AV node level- inferior wall MI, digoxin and rheumatic fever. At bundle branch level- anterior wall MI. Chronic- Commonest cause- fibrosis (Lev, Lenegre) Congenital (maternal anti-Ro antibodies) Chronic third degree HB is usually due to damage to both right and left [...]]]></description>
			<content:encoded><![CDATA[<p><strong>Third degree AV block</strong></p>
<ul>
<li>May be      isoarrhythmic- similar P and QRS rates.</li>
<li>Acute-
<ul>
<li>At AV       node level- inferior wall MI, digoxin and rheumatic fever.</li>
<li>At       bundle branch level- anterior wall MI.</li>
</ul>
</li>
<li>Chronic-
<ul>
<li>Commonest       cause- fibrosis (Lev, Lenegre)</li>
<li>Congenital       (maternal anti-Ro antibodies)</li>
</ul>
</li>
<li>Chronic      third degree HB is usually due to damage to both right and left bundle      branches.</li>
<li>When      difficulty in distinguishing third degree HB from second degree HB-
<ul>
<li>QRS       intervals are usually regular- if irregular usually suggests some amount       of AV conduction.</li>
<li>Constant       PR (or flutter-R) interval indicates second degree HB while varying PR       (or flutter-R) interval indicates third degree HB.</li>
</ul>
</li>
</ul>
<p><strong>Location of AV block</strong></p>
<ul>
<li>Sites-
<ul>
<li>AV       node- most common</li>
<li>His       bundle- rare (so differentiation is between the other two)</li>
<li>Bundle       branches- second most common</li>
</ul>
</li>
<li>With      blocks lower than third degree heart blocks-
<ul>
<li>QRS       duration-
<ul>
<li>Normal-        AV node</li>
<li>Wide-        can be either (AV node + aberrancy can also cause widening)</li>
</ul>
</li>
<li>PR       interval consistency-
<ul>
<li>Variable        PR interval- AV node (only AV node has ability to vary conduction time)</li>
<li>Constant        PR interval (when AV conduction is present)- bundle branch</li>
</ul>
</li>
</ul>
</li>
</ul>
<p><strong>Wenckebach sequence</strong></p>
<ul>
<li>
<ul>
<li>Indicates       AV nodal block</li>
<li>PR       interval-
<ul>
<li>Progressive        prolongation of PR interval &#8211; because successive atrial impulses arrive        progressively earlier in the relative refractory period of the AV node.</li>
<li>The        “increase in PR interval” (PR2-PR1, PR3-PR2 etc) progressively decreases        and hence is maximum between the second beat and the first beat. This,        in the setting of a constant PP interval, causes RR interval to shorten        progressively.</li>
</ul>
</li>
<li>RR       interval-
<ul>
<li>RR interval        progressively shortens.</li>
<li>Longest        cycle (with the missed beat) is less than twice the shortest one. This        is because the longest cycle has the shortest PR interval beat.</li>
<li>Smaller        QRS groups are commoner than larger ones. Thus, 3:2 &gt; 4:3 &gt; 5:4        etc. Commonest is pairs.</li>
</ul>
</li>
<li> PP interval- constant.</li>
</ul>
</li>
</ul>
<p><strong>Purkinje block (in bundle branches)</strong></p>
<ul>
<li>
<ul>
<li>Second       degree heart block-
<ul>
<li>Purkinje        cells have very short refractory period. So, either normal PR or no        conduction. No increased PR interval.</li>
</ul>
</li>
<li>Complete       heart block-
<ul>
<li>Almost        always, preceding bundle branch block is present.</li>
<li>Usually        there is sudden transition from no heart block to complete heart block        without an intervening first or second degree heart block.</li>
</ul>
</li>
</ul>
</li>
</ul>
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		<item>
		<title>ECG- Decreased automaticity leading to bradycardia</title>
		<link>http://www.heartpearls.com/2009/07/ecg-decreased-automaticity-leading-to-bradycardia.html</link>
		<comments>http://www.heartpearls.com/2009/07/ecg-decreased-automaticity-leading-to-bradycardia.html#comments</comments>
		<pubDate>Wed, 22 Jul 2009 11:47:43 +0000</pubDate>
		<dc:creator>Dr Jayachandran Thejus MD</dc:creator>
				<category><![CDATA[ECG]]></category>

		<guid isPermaLink="false">http://www.heartpearls.com/?p=452</guid>
		<description><![CDATA[Physiologic slowing of sinus node- sinus bradycardia Atheletes Expiration Pathologic slowing of sinus node- Sick sinus syndrome SA block Physiologic or pathologic parasympathetic activation- sinus bradycardia and AV block Increased intracranial or intraocular pressure Vasovagal reflex, carotid reflex Venodilators]]></description>
			<content:encoded><![CDATA[<ul>
<li>Physiologic      slowing of sinus node- sinus bradycardia
<ul>
<li>Atheletes</li>
<li>Expiration</li>
</ul>
</li>
<li>Pathologic      slowing of sinus node-
<ul>
<li>Sick       sinus syndrome</li>
<li>SA       block</li>
</ul>
</li>
<li>Physiologic      or pathologic parasympathetic activation- sinus bradycardia and AV block
<ul>
<li>Increased       intracranial or intraocular pressure</li>
<li>Vasovagal       reflex, carotid reflex</li>
<li>Venodilators</li>
</ul>
</li>
</ul>
]]></content:encoded>
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		<item>
		<title>ECG- Ventricular tachyarrhythmias</title>
		<link>http://www.heartpearls.com/2009/07/ecg-re-entrant-ventricular-tachyarrhythmias.html</link>
		<comments>http://www.heartpearls.com/2009/07/ecg-re-entrant-ventricular-tachyarrhythmias.html#comments</comments>
		<pubDate>Tue, 21 Jul 2009 17:56:01 +0000</pubDate>
		<dc:creator>Dr Jayachandran Thejus MD</dc:creator>
				<category><![CDATA[ECG]]></category>

		<guid isPermaLink="false">http://www.heartpearls.com/?p=446</guid>
		<description><![CDATA[Usual VT is due to reentry and has rate more than 120/mt. AIVR is due to accelerated automaticity and has rate less than 120/mt. Torsades de pointes is due to macro-reentry. Etiologies Idiopathic- RVOT, fascicular CAD- acute MI, past MI Dilated cardiomyopathy, hypertrophic cardiomyopathy ARVC Drugs Diagnosis Step 1- search the whole ECG for P [...]]]></description>
			<content:encoded><![CDATA[<ul>
<li>Usual VT      is due to reentry and has rate more than 120/mt. AIVR is due to      accelerated automaticity and has rate less than 120/mt.</li>
<li>Torsades      de pointes is due to macro-reentry.</li>
</ul>
<p><strong>Etiologies</strong></p>
<ul>
<li>Idiopathic-      RVOT, fascicular</li>
<li>CAD-      acute MI, past MI</li>
<li>Dilated      cardiomyopathy, hypertrophic cardiomyopathy</li>
<li>ARVC</li>
<li>Drugs</li>
</ul>
<p><strong>Diagnosis</strong></p>
<ul>
<li>Step      1- search the whole ECG for <strong>P waves</strong>
<ul>
<li>AV       dissociation
<ul>
<li>Especially        likely to be seen in leads with low QRS amplitude and in lead v1 (where        P waves tend to be prominent)</li>
<li>May        be absent if there is retrograde atrial activation</li>
<li>Entirely        specific, but poorly sensitive</li>
</ul>
</li>
<li>VA       association
<ul>
<li>May        be 1:1 or lower.</li>
</ul>
</li>
</ul>
</li>
<li>Step      2- search the whole ECG for <strong>fusion      or capture beats</strong>
<ul>
<li>Fusion       or capture beats. The latter is premature.</li>
<li>Seen       only at low rates of less than 160/mt.</li>
</ul>
</li>
<li>Step 3-      search the <strong>precordial leads for RS</strong> pattern
<ul>
<li>No       RS pattern in any precordial lead
<ul>
<li>Strongly        suggests VT. (With RBBB or LBBB, there will be RS in at least one lead.)</li>
</ul>
</li>
<li>RS       is present in a precordial lead
<ul>
<li>It can        still be VT if time from beginning of R to nadir of S is more than 100        msec.</li>
</ul>
</li>
</ul>
</li>
<li>Step 4-      study <strong>QRS morphology in precordial      leads</strong>
<ul>
<li>RBBB       pattern
<ul>
<li>V1-
<ul>
<li>R         (can also occur with RVH or PWMI)</li>
<li>R         with notched downslope producing two rabbit ears, first being taller (Marriott         sign, very specific)</li>
<li>QR         (can also occur with RBBB + MI)</li>
</ul>
</li>
<li>V6-
<ul>
<li>QS</li>
<li>rS         + left axis deviation</li>
</ul>
</li>
</ul>
</li>
<li>LBBB       pattern
<ul>
<li>V1-
<ul>
<li>R         duration 40 msec or more</li>
<li>Notched         downslope in S</li>
<li>Onset         of R to nadir of S 60 msec or more</li>
</ul>
</li>
<li>V6-
<ul>
<li>QS</li>
<li>qR</li>
</ul>
</li>
</ul>
</li>
</ul>
</li>
</ul>
<p><strong>Other interesting points</strong></p>
<ul>
<li>
<ul>
<li>RV       origin VT occurs in young females while LV origin VT occurs in elderly males       with heart disease.</li>
<li>TDP       is usually nonsustained.</li>
<li>Ventricular       flutter and fibrillation are due to macro-reentry.</li>
<li>Ventricular       fibrillation is more difficult to defibrillate than ventricular flutter.</li>
</ul>
</li>
</ul>
]]></content:encoded>
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		</item>
		<item>
		<title>ECG- Supraventricular tachycardia with aberrancy- features to distinguish from ventricular tachycardia</title>
		<link>http://www.heartpearls.com/2009/06/what-are-the-main-features-of-supraventricular-tachycardia-with-aberrancy-that-helps-to-distinguish-if-from-ventricular-tachycardia.html</link>
		<comments>http://www.heartpearls.com/2009/06/what-are-the-main-features-of-supraventricular-tachycardia-with-aberrancy-that-helps-to-distinguish-if-from-ventricular-tachycardia.html#comments</comments>
		<pubDate>Tue, 02 Jun 2009 13:53:21 +0000</pubDate>
		<dc:creator>Dr Jayachandran Thejus MD</dc:creator>
				<category><![CDATA[ECG]]></category>
		<category><![CDATA[aberrancy]]></category>
		<category><![CDATA[supraventricular tachycardia]]></category>
		<category><![CDATA[Ventricular tachycardia]]></category>

		<guid isPermaLink="false">http://www.heartpearls.com/?p=197</guid>
		<description><![CDATA[Distinguishing supraventricular tachycardia from aberrancy from ventricular tachycardia is very important in routine clinical practice.]]></description>
			<content:encoded><![CDATA[<ol type="1">
<li class="MsoNormal">RBBB      pattern (rsR’ in v1 or qrS in v6).</li>
<li class="MsoNormal">Initial      part of the complex is similar to a sinus rhythm complex.</li>
<li class="MsoNormal">Similar      complexes seen earlier when patient was in sinus rhythm and then was      clearly diagnosed as aberrant conduction.</li>
<li class="MsoNormal">Complex      ending a short RR interval after a long RR interval (preceding long RR-      short RR sequence; second in a row phenomenon; Ashman phenomenon).</li>
<li class="MsoNormal">The      suspected complexes are all having preceding P waves.</li>
<li class="MsoNormal">RBBB      and LBBB patterns seen- more likely to be alternating BBB due to varying      aberrancy rather than ventricular tachycardias from both ventricles.</li>
</ol>
]]></content:encoded>
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