• Past MI causes monomorphic VT.
  • Acute ischemia causes polymorphic VT.
  • Bundle branch reentrant VT can be treated by ablating the right bundle branch.
  • ARVC VT- LBBB pattern VT.
  • ECG findings in ARVC-
    • RBBB
    • T inversion in v1 to v3
    • Epsilon wave- notch in terminal part of QRS.
  • ARVC is more in males.
  • CPVT is first bidirectional and then polymorphic.
  • CPVT is highly reproducible.
  • In Brugada syndrome, sodium channels are non-functional or have accelerated recovery.
  • Torsades de pointes was originally described in bradycardia due to complete heart block.
  • TDP is initiated by a late PVC falling on the termination of a long T wave.
  • Short coupled variant of TDP is initiated by an early PVC. It has high mortality.
  • TDP is initiated by early afterdepolarizations and perpetuated by reentry.
  • Jervell-Lange-Nielsen syndrome is named after two persons- Jervell and Lange-Nielsen.
  • In LQT3, arrhythmias occur during sleep.
  • Stress testing provokes long QT and ventricular arrhythmias in LQTS.
  • LVOT VT is distinguished from RVOT VT by S in lead I and transition zone between v1 & v2.
  • RVOT VT is terminated by adenosine while fascicular VT is not. Both are terminated by verapamil.
  • Causes of bidirectional VT are digoxin toxicity and CPVT.
  • BB reentrant VT usually has LBBB pattern.
  • VF is more in morning while asystole is not.
  • Resuscitated VF has less chance of recurrence of VF if MI evolves.